Author Correction: TLR9 activation via microglial glucocorticoid ...

4 downloads 0 Views 264KB Size Report
Jun 22, 2018 - 1 Institute of Biology Paris Seine, Gene Regulation and Adaptive ... CEA, Université Paris-Sud, Université Paris-Saclay (UMR9199), F-92265 ...
DOI: 10.1038/s41467-018-05680-w

OPEN

Author Correction: TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons 1234567890():,;

Layal Maatouk1, Anne-Claire Compagnion1, Maria-Angeles Carrillo-de Sauvage2, Alexis-Pierre Bemelmans Sabrina Leclere-Turbant3, Vincent Cirotteau1, Mira Tohme4, Allen Beke1, Michaël Trichet5, Virginie Bazin5, Bobby N. Trawick6, Richard M. Ransohoff7, François Tronche1, Bénédicte Manoury4 & Sheela Vyas1

2,

Correction to: Nature Communications ; https://doi.org/10.1038/s41467-018-04569-y; published online 22 June 2018 The originally published version of this Article contained an error in the subheading “Microglial GR does not affect DN loss triggered by TLR4 and TLR7”, which was incorrectly given as “Microglial GR does affect DN loss triggered by TLR2 and TLR4.” This has now been corrected in both the PDF and HTML versions of the Article.

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. © The Author(s) 2018

1 Institute

of Biology Paris Seine, Gene Regulation and Adaptive Behaviors Team, Department of Neuroscience Paris Seine, Sorbonne Université, CNRS UMR 8246 & INSERM U1130, 9 Quai Saint Bernard, F-75005 Paris, France. 2 CEA, DRF, Institut François Jacob, Neurodegenerative Diseases Laboratory, Molecular Imaging Research Center (MIRCen), CNRS, CEA, Université Paris-Sud, Université Paris-Saclay (UMR9199), F-92265 Fontenay-aux-Roses, France. 3 IHU-AICM–Neuro-CEB, Plateforme de Ressources Biologiques (PRB), Hôpital de la Pitié-Salpétrière, 47 Boulevard de l’Hôpital, F-75013 Paris, France. 4 INEM, INSERM U1151-CNRS UMR 8253, Hôpital Necker, Université Paris Descartes, Sorbonne Paris Cité, Faculté de Médecine, 149 Rue de Sèvres, F-75005 Paris, France. 5 Institute of Biology Paris Seine, Electron Microscopy Facility, Sorbonne Université FR3631, 9 Quai Saint Bernard, F-75005 Paris, France. 6 Center for Organic Chemistry, Mallinckrodt Pharmaceuticals, 3600N. Second Street, B81-T l, 63147 St. Louis, MO, USA. 7 Third Rock Ventures, 02116 Boston, MA, USA. Correspondence and requests for materials should be addressed to S.V. (email: [email protected]) NATURE COMMUNICATIONS | (2018)9:3109 | DOI: 10.1038/s41467-018-05680-w | www.nature.com/naturecommunications

1