Severe Coronary Vasospasm Complicated with Ventricular Tachycardia
Recommend Documents
Feb 18, 2016 - cardiac involvement may include transient myocardial dysfunction, ... myocardial ischemia or severe coronary vasospasm by the excessive.
triggered by stress or exertion. This review ... polymorphic ventricular tachycardia: An exciting new era. .... foci of triggered activity in the right and left ventricles,.
Aug 4, 2010 - Spontaneous severe multivessel coronary artery vasospasm is a rare but .... The pathophysiological mechanism of vasospastic angina.
Jun 14, 2014 - described having coronary vasospasm postpartum and even .... spasm has been associated with patients with vasospastic angina. Park et al.
Feb 13, 2018 - tachycardia (VT) in coronary artery disease (CAD): A study from tertiary care center ... status, treatment received and finally the outcome of CAD patients manifesting as sustained VT. ..... Steg PG, James SK, Atar D, Badano LP,.
Postgraduate Medical Journal (February 1984) 60, 155-156. Fatal ventricular tachycardia in association with propafenone, a new class IC antiarrhythmic agent.
institute, because of a complete atrioventricular (AV) block ... by 18 ms, at the proximal area of the left posterior fascicle. (site C in .... the basal ventricular septum.
Jul 19, 2018 - After an inpatient investigative evaluation, sotalol was trialled with ... had initiated Sotalol for its combined beta blockage and Class III.
Mar 19, 2018 - ognized to properly treat the patient, as verapamil treatment is effective. Keywords. African, Belhassen tachycardia, right axis, right bundle branch block, verapamil ... the slow conduction of calcium in partial depolarization. [9].
Ventricular tachycardia (VT) is frequently the precursor of ventricular fibril- lation. Therefore, preventive treatment of VT is a major goal to reduce sud- den death.
ventricular fascicles.2 The incidence of VT originating from mitral annulus has been reported as ... block (RBBB), left superior axis, and negative QRS concordance in precordial leads .... the LV to the basal septum as directly or indirectly having.
incidence of unexplained sudden death (Alderman &. Coltart, 1982; Friedman et al., 1982). Sincemyocar- dial ischaemia, both acutely and overa long period of.
Severe Coronary Vasospasm Complicated with Ventricular Tachycardia
Introduction. Variant form of angina was first described by Prinzmetal as transient and recurrent coronary spasm leading to recurrent episodes of myocardial ...
Case Report Severe Coronary Vasospasm Complicated with Ventricular Tachycardia Göksel Acar, Serdar Fidan, Servet İzci and Anıl Avcı Kartal Koşuyolu High Specialty Education and Research Hospital, Cardiology Department, Istambul - Turkey
Introduction Variant form of angina was first described by Prinzmetal as transient and recurrent coronary spasm leading to recurrent episodes of myocardial ischemia1. Although so many factors can trigger or aggravate coronary vasospasm; endothelial dysfunction was considered as an important mechanism underlying this situation. The location and extent of vasospasm determine the seriousness of the clinical picture, which can include myocardial infarction severe arrhythmias, cardiogenic shock and even death2.
Case Report A 68-year-old male was admitted to our emergency department complaining of severe chest pain for 4 hours. On physical examination his vital findings were within the normal limits with a slightly high blood pressure reading 150/90 mmHg, a regular pulse 80/min, a respiratory rate 15/min. His 12-lead electrocardiogram (ECG) showed 1mm ST depression in lateral leads without significant ST elevation or T wave abnormality in other leads. The patient had a history of hypertension and a coronary angiography which was performed 10 days before in another hospital. The patient reported that he had had similar type of chest pain 10 days before and because of that a coronary angiography had been performed. There was not any critical lesion on his coronary angiogrpahy and he had been discharged on metoprolol 50mg and aspirin 100mg because of noncritical coronary lesions (Figure 1A, 1B). A bed side transthoracic echocardiogram revealed posterolateral wall hypokinesis with an ejection fraction around 50-55 %. The patient's cardiac enzyme levels on admission were above normal with a troponin T level 4.2 ng/ml (
